Figure 3
Figure 3. Effects of lysosome and proteasome inhibitors on TpoR cell-surface and total levels in JAK2 V617F cells. (A) Flow cytometric assessment of cell-surface HA-TpoR levels of Ba/F3 TpoR JAK2 V617F treated or not with the indicated inhibitors. Shown are averages of at least 3 independent experiments ± SD. Each experiment reflected analysis of 10 000 cells. **Two-tailed P < .01. (B) The indicated Ba/F3 cells were treated with 20 μg/mL cycloheximide for 20 hours or not and simultaneously treated with a combination of lysosome inhibitors (10μM, E64, 10μM pepstatin A, and 200μM leupeptin) or 10μM MG132 proteasome inhibitor. Shown are Western blot analyses for HA-TpoR and β-actin. (C) Western blot analysis with anti-HA antibodies and anti–β-actin antibodies of Ba/F3 TpoR JAK2 V617F cells treated with the indicated inhibitors for 20 hours. The densitometric ratios between the mature and immature TpoR bands were normalized to that of untreated cells.

Effects of lysosome and proteasome inhibitors on TpoR cell-surface and total levels in JAK2 V617F cells. (A) Flow cytometric assessment of cell-surface HA-TpoR levels of Ba/F3 TpoR JAK2 V617F treated or not with the indicated inhibitors. Shown are averages of at least 3 independent experiments ± SD. Each experiment reflected analysis of 10 000 cells. **Two-tailed P < .01. (B) The indicated Ba/F3 cells were treated with 20 μg/mL cycloheximide for 20 hours or not and simultaneously treated with a combination of lysosome inhibitors (10μM, E64, 10μM pepstatin A, and 200μM leupeptin) or 10μM MG132 proteasome inhibitor. Shown are Western blot analyses for HA-TpoR and β-actin. (C) Western blot analysis with anti-HA antibodies and anti–β-actin antibodies of Ba/F3 TpoR JAK2 V617F cells treated with the indicated inhibitors for 20 hours. The densitometric ratios between the mature and immature TpoR bands were normalized to that of untreated cells.

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