Table 3.

Recommendations for diagnostic workup and treatment of CAR T-cell–related myelopathy

We recommendTo be consideredInvestigational
Diagnosis Brain and spine MRI with and without contrast
Evaluation for constipation and urinary retention with bladder scan
Early consultations with neurology and infectious disease specialists
If no contraindication, lumbar puncture with opening pressure measurement and CSF analysis (biochemistry, cell count, cytology, flow cytometry, and oligoclonal bands)
Infectious workup on CSF, including cultures and NAT for HHV-6, CMV, VZV, HSV-1/2, JC-virus, EBV, adenovirus, and enteroviruses
Infectious workup on peripheral blood, including cultures, QuantiFERON, HIV and hepatitis testing, Treponema pallidum testing, NAT for HHV-6, EBV, adenovirus, and CMV
Serologic investigation, including vitamin B12, folate, methylmalonic acid, homocysteine, vitamin E, copper, ceruloplasmin, ferritin, C-reactive protein, CBC, LDH, liver enzymes, bilirubin, INR, PTT, creatinine, and extended electrolytes 
Autoantibody testing (onconeural, AQP4, MOG antibodies) in CSF if previous exposure to ICIs and as per neurology for other patients
Extended infectious workup on CSF and peripheral blood guided by an infectious disease specialist Serologic investigations if suspicion of IEC-HS, including triglycerides, fibrinogen, lactate, and soluble IL-2 receptor-α.
Other serologic test: thiamine level
Serology analysis for rheumatoid disease and NMO, including ANA, anti-Ro/La, anti-AQP4 testing, and other autoantibodies as guided by the clinical picture
Electrodiagnostic studies 
Extended cytokine panel on peripheral blood and/or CSF
Lymphocytes subsets in peripheral blood and/or CSF
Measurement of CAR T-cell expansion in peripheral blood and/or CSF 
Treatment High-dose steroids: methylprednisolone 1 g IV daily for 3-5 d with a slow taper depending on response and etiologic workup
Avoidance of tocilizumab if no concurrent CRS
If an infectious cause is identified: targeted antimicrobial therapy
Medication review and avoidance of neurotoxic agents
Correction of deficiencies in essential micronutrients
Correction of hyponatremia
Early rehabilitation 
Empirical thiamine repletion: for example, 500 mg every 8 h for 72 h, followed by daily maintenance supplementation
Empirical treatment for HHV-6 and CMV while awaiting results
Other empirical infectious treatments if there are risk factors and if patient is not improving on initial steroid therapy
IVIG 2 g/kg administered in divided doses per package insert, or plasmapheresis if previous exposure to ICIs. IVIG could be considered for other patients if refractory to initial treatment with pulse dose steroids
Anakinra if refractory to initial management 
Siltuximab
Emapalumab
Canakinumab
Basiliximab
Ruxolitinib
BTK inhibitors
Other TKI
Salvage therapy (etoposide, cyclophosphamide, alemtuzumab, and ATG) 
We recommendTo be consideredInvestigational
Diagnosis Brain and spine MRI with and without contrast
Evaluation for constipation and urinary retention with bladder scan
Early consultations with neurology and infectious disease specialists
If no contraindication, lumbar puncture with opening pressure measurement and CSF analysis (biochemistry, cell count, cytology, flow cytometry, and oligoclonal bands)
Infectious workup on CSF, including cultures and NAT for HHV-6, CMV, VZV, HSV-1/2, JC-virus, EBV, adenovirus, and enteroviruses
Infectious workup on peripheral blood, including cultures, QuantiFERON, HIV and hepatitis testing, Treponema pallidum testing, NAT for HHV-6, EBV, adenovirus, and CMV
Serologic investigation, including vitamin B12, folate, methylmalonic acid, homocysteine, vitamin E, copper, ceruloplasmin, ferritin, C-reactive protein, CBC, LDH, liver enzymes, bilirubin, INR, PTT, creatinine, and extended electrolytes 
Autoantibody testing (onconeural, AQP4, MOG antibodies) in CSF if previous exposure to ICIs and as per neurology for other patients
Extended infectious workup on CSF and peripheral blood guided by an infectious disease specialist Serologic investigations if suspicion of IEC-HS, including triglycerides, fibrinogen, lactate, and soluble IL-2 receptor-α.
Other serologic test: thiamine level
Serology analysis for rheumatoid disease and NMO, including ANA, anti-Ro/La, anti-AQP4 testing, and other autoantibodies as guided by the clinical picture
Electrodiagnostic studies 
Extended cytokine panel on peripheral blood and/or CSF
Lymphocytes subsets in peripheral blood and/or CSF
Measurement of CAR T-cell expansion in peripheral blood and/or CSF 
Treatment High-dose steroids: methylprednisolone 1 g IV daily for 3-5 d with a slow taper depending on response and etiologic workup
Avoidance of tocilizumab if no concurrent CRS
If an infectious cause is identified: targeted antimicrobial therapy
Medication review and avoidance of neurotoxic agents
Correction of deficiencies in essential micronutrients
Correction of hyponatremia
Early rehabilitation 
Empirical thiamine repletion: for example, 500 mg every 8 h for 72 h, followed by daily maintenance supplementation
Empirical treatment for HHV-6 and CMV while awaiting results
Other empirical infectious treatments if there are risk factors and if patient is not improving on initial steroid therapy
IVIG 2 g/kg administered in divided doses per package insert, or plasmapheresis if previous exposure to ICIs. IVIG could be considered for other patients if refractory to initial treatment with pulse dose steroids
Anakinra if refractory to initial management 
Siltuximab
Emapalumab
Canakinumab
Basiliximab
Ruxolitinib
BTK inhibitors
Other TKI
Salvage therapy (etoposide, cyclophosphamide, alemtuzumab, and ATG) 

ANA, antinuclear antibody; AQP4, aquaporin-4; ATG, antithymocyte globulin; BTK, Bruton’s tyrosine kinase; CBC, complete blood count; HSV-1/2, herpes simplex virus 1/2; INR, international normalized ratio; LDH, lactate dehydrogenase; MOG, myelin oligodendrocyte glycoprotein; NAT, nucleic acid testing; NMO, neuromyelitis optica; PTT, partial thromboplastin time; TKI, tyrosine kinase inhibitor; VZV, varicella-zoster virus.

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